Fibromyalgia
Fibromyalgia is not in your head. The pain is real, the fatigue is real, and the cognitive impairment is real. What's missing isn't validation — it's investigation. Conventional medicine labels fibromyalgia when it can't find the cause. We keep looking.
Fibromyalgia involves central sensitization, mitochondrial dysfunction, neuroinflammation, and autonomic nervous system dysregulation — not 'imaginary pain.' Signal-Based Medicine™ evaluates the objective markers behind the pain: mitochondrial energy production, inflammatory cytokines, small fiber nerve function, and the gut-immune-neurological axis that drives sensitization.
Symptoms
- Widespread body pain
- Fatigue
- Brain fog ('fibro fog')
- Sleep disruption
- Headaches
- IBS symptoms
- Sensitivity to touch, light, sound
- Morning stiffness
- Numbness and tingling
- Temperature sensitivity
Root Causes
Fibromyalgia is a central sensitization disorder — your nervous system has turned up the volume on pain signals. But why? Mitochondrial dysfunction means nerves lack energy to function properly. Neuroinflammation sensitizes pain pathways. Gut dysfunction drives systemic inflammation. Small fiber neuropathy (now found in many fibromyalgia members) is a measurable nerve disorder. These are testable, treatable findings — not mysteries.
Testing
- Inflammatory cytokine panel
- Mitochondrial function markers
- Organic acids test
- Thyroid panel
- GI-MAP
- Small fiber neuropathy evaluation
- KureBioMap™ assessment
Treatment Approach
We address the objective mechanisms behind fibromyalgia: mitochondrial support, neuroinflammation reduction, gut restoration, HPA axis repair, and nervous system calming — systematically, based on your KureBioMap™ findings.
Frequently Asked Questions
Is fibromyalgia all in my head? No. Research has identified measurable biological findings in fibromyalgia including small fiber neuropathy, elevated inflammatory cytokines, mitochondrial dysfunction, and autonomic nervous system dysregulation. The pain is real and has biological causes.
Why don't pain medications work well for fibromyalgia? Because the problem isn't in the tissues — it's in the nervous system processing of pain signals. Pain medications target peripheral pain, not central sensitization. Addressing the underlying drivers (inflammation, mitochondrial function, gut health) is more effective.
Can fibromyalgia improve? Yes. Many members experience significant improvement when the underlying biological drivers — mitochondrial dysfunction, neuroinflammation, gut dysbiosis, hormonal imbalance — are identified and addressed through Signal-Based Medicine.
